Mitochondria are altered in human cell model of Parkinson's disease

Based on research in fruit flies, it has long been suspected that the most common mutation linked to both sporadic and familial Parkinson's disease wreaks its havoc by altering the function of mitochondria in neurons that produce the neurotransmitter dopamine. Using stem cells derived from patients who have PD, scientist at the Buck Institute have confirmed that finding in human cells for the first time.

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