Study reveals possible treatment for diseases caused by Mitofusin 2 deficiency

In mice, mitochondria in healthy and Mfn1 -deficient heart muscle cells are organized in a linear arrangement, but the organelles are enlarged and disorganized in Mfn2 -deficient cells . Credit: Mourier et al., 2015 Researchers have discovered a novel role for Mitofusin 2, and the findings may point to a new treatment for patients with diseases caused by loss of the mitochondrial protein.

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